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The Orewa Speech: another threat to Maori health?
Cindy Towns, Nathan Watkins, Arapera Salter, Patricia Boyd,
Lianne Parkin
On the 27th of January,
2004, Don Brash addressed the Orewa Rotary Club on the subject of race
relations, and what he perceived to be the preferential treatment of Maori in
health and
education.1
According to the New Zealand Herald newspaper, ‘The Orewa Speech’
has had a ‘seismic impact on the political landscape’ in New
Zealand.2
The National Party’s meteoric rise in the
polls3
suggests that their new leader has indeed struck a chord with the New Zealand
public.
Brash
established his stance early in the speech by stating:
‘We
are one country with many peoples, not simply a society of Maori and Pakeha
where the minority has a birthright to the upper hand’.
He
later claimed:
‘In
both education and healthcare, government funding is now influenced not just by
need—as it should be—but also by the ethnicity of the
recipient.1
This viewpoint article seeks to evaluate, from the
perspective of health, whether specifically addressing the needs of Maori is
justified—or whether (as Brash suggests) such an approach cannot be
justified. One way to determine the legitimacy of a targeted strategy, is to
prove that Maori have a need in excess of other societal groups. With regard to
need, mortality and life expectancy data are arguably the most frequently used
measures of health status. This is due, in part, to their accessibility;
certification of death being a legal requirement in most industrialised
countries.4
In New Zealand, the most recent life expectancy figures (2000–2002) show
that non-Maori live on average 8.5 years longer than
Maori.5
Specifically, life expectancy at birth for females of Maori ethnicity is 73.2
years compared with 81.9 years for their non-Maori counterparts. For men, life
expectancy at birth is 69.0 for Maori and 77.2 years for non-Maori.
Until recently, meaningful comparisons of ethnic-specific
mortality rates and the study of time trends in Maori and Pacific mortality
rates have been hampered by changing definitions of ethnicity and a
numerator-denominator
bias. 6
Mortality rates are calculated using two primary sources: death registration
forms (the numerator) and census records (the denominator). Between 1981 and
1995 funeral directors completing deaths registration forms used a biological
definition of ethnicity (half or more blood), while the census collected
self-identified ethnicity data. This resulted in an undercounting of Maori and
Pacific deaths and the underestimation of Maori and Pacific mortality rates.
Ajwani et al (2003) address this bias in their comprehensive
description of trends in ethnic-specific mortality rates in New Zealand from
1980
to1999.6
Their results are disturbing. There has been little, if any, decline in Maori
and Pacific mortality rates over the last two decades, despite a steady decline
in non-Maori non-Pacific rates. Hence, the gaps in life expectancy between these
groups increased markedly over the 1980s and 1990s.
The major contribution to the disparity has been the
differential chronic disease mortality in the middle to older age
groups—most notably from ischaemic heart disease, diabetes, and cancer.
For example, for the prioritised series (those persons identifying Maori
ethnicity as their primary, but not sole, ethnic group) age-standardised cancer
mortality rates were 2.0 (male) and 2.1 (female) times greater for Maori than
for non-Maori non-Pacific people during 1996–1999.
Cardiovascular mortality rates have decreased over the last
two decades, but to a lesser extent amongst Maori. Consequently, by
1996–99, cardiovascular mortality rates were 3.0 times higher for Maori
males compared to non-Maori non-Pacific males, and 4.2 times higher for Maori
females compared to non-Maori, non-Pacific
females.6
Carr et al (2002) found similar disparities in a study of heart failure
outcomes.7
In a retrospective analysis, they compared outcomes for Maori and non-Maori
using mortality and hospital admission data from 1988 to 1998. Mortality from
heart failure was more than 8.8 times higher among Maori men aged 45–64
years when compared to non-Maori.
To understand what is contributing to these disparities in
life expectancy and mortality rates, it is necessary to consider some of the
determinants of health status. It has long been recognised, for example, that
poor socioeconomic status is associated with poor health outcomes. In fact, the
link was established as early as 1840, when reliable data first became
available.8
More recently, it has been observed that about 80% of the 80
most-common causes of death occur more frequently in lower socioeconomic
groups.9
Ajwani et al (2003) also cite numerous data from both the United Kingdom and
United States that confirm that widening health inequalities are strongly
associated with widening socioeconomic
inequalities.6
Similar gradients are evident within the New Zealand population.
Several tools are available for measuring socioeconomic
status. The NZDep96 is an area-based measure of socioeconomic deprivation that
uses nine variables obtained from 1996 census data to provide a summary
deprivation score for each meshblock (a geographical unit containing a median of
90 people) in New Zealand. The score is based on the proportion of people within
each meshblock who don’t have access to a telephone or car, are
unemployed, in receipt of a means-tested income, live in a low income or
single-parent family, have no educational qualifications, live in non-tenured
homes, and live in crowded households. The meshblocks are ranked into deciles
with one being the least deprived and 10 being the most
deprived.10
Although care should be taken in using NZDep96 as the sole
indicator of socioeconomic status, the ranking of individuals by the decile
assigned to their neighbourhood is, on average, strongly and linearly related to
health and other social
outcomes.11
An examination of NZDep96 by ethnicity reveals that the Maori ethnic group is
over-represented in the most deprived areas. Reid et al (2000) found that more
than half (56%) of Maori live in areas represented by the three lowest
deciles.12
Indeed, only 3% of Maori are represented in decile one (the
most affluent) whilst 26% reside in decile 10 (the most deprived). This pattern
is consistent within both categories of the Maori ethnic group: sole Maori
(those who give Maori as their only ethnicity) and mixed Maori (those who give
Maori as only one of their ethnicities). Other socioeconomic data reveal that
Maori households (when compared with non-Maori) earn on average $10,000 less
than non-Maori households, are more likely to rely on income support than
non-Maori, and are more likely to run out of food because of a lack of
money.13
Hence, these data suggest that Maori are over-represented in
the lower socioeconomic classes, and that this level of deprivation puts them at
increased risk of poor health outcomes. However, there is clear evidence that
poor health outcomes in Maori are not simply due to socioeconomic deprivation.
For example, Reid et al (2000) combined NZDep96 data from deciles 1–7 (due
to the relatively few number of Maori in this category) and deciles 8–9,
and compared them with decile
10.12
They found that life expectancy of Maori from birth is consistently less than
that of non-Maori at each of these three deprivation points. These authors state
that such data provide support for the argument that ‘above and beyond the
effects of increasing deprivation in New Zealand, there is an additional and
significant health effect of ethnicity’.
Similar patterns are noted by Sporle et al (2002), who used
data from 1996–97 to update previous studies of social-class mortality
differences in Maori and non-Maori New Zealand men aged 15–64
years.14
They found that within each social class, Maori all-cause mortality was
significantly higher than non-Maori, and that these disparities increased with
increasing disadvantage. Moreover, the mortality rate of Maori in the highest
social class was still higher than that of non-Maori in the lowest social class.
The greatest disparities between Maori and non-Maori were found in an analysis
of amenable causes of death, that is, deaths that should have been avoidable had
the individuals received appropriate medical care.
Sporle and colleagues conclude that the persistently high
Maori mortality rates, when controlled for social class, indicate that the poor
state of Maori health cannot be explained solely by relative socioeconomic
disadvantage. They further note that the high Maori rate of potentially
preventable deaths indicates that the health sector is still not meeting the
serious health needs of this ethnic group. Indeed, in terms of targeting health
resources, this research indicates that Maori ethnicity and high need are
synonymous.
In relation to other determinants of health, there is
evidence to suggest that Maori are over-represented among those with
intermediate risk factors for disease. Provisional results of the 2003/03 New
Zealand Health Survey demonstrate that the prevalence of smoking in Maori is
more than twice that of non-Maori, and higher than any other ethnic
group.15
This data also shows that the prevalence of hypertension (a major risk factor
for heart failure) is highest in Maori and that approximately 30% of Maori are
obese. These risk factors have a considerable impact on both the rate and
outcomes of amenable disease and are likely to contribute to the significant
burden of disease suffered by Maori.
Disparities between Maori and non-Maori in the treatment of
chronic disease have also been observed. For example, diabetic Maori in one
study appeared to have poor glycaemic control and poor knowledge about
diabetes.16
The South Auckland Diabetes Study confirmed these
findings17
whilst other research demonstrates that Maori patients are more likely to have
diabetic complications such as nephropathy and
retinopathy.18
Research into cardiovascular disease and outcomes suggests
that, despite high hospitalisation rates for cardiac diseases, intervention
rates for Maori are much
lower.19
Similarly, Tukuitonga and Bindman (2002) state that even though Pacific and
Maori peoples have higher rates of coronary artery disease morbidity and
mortality, revascularisation rates are lower in both
groups.20
Adequate access to care is also raised by Ellison-Loschmann et al (2004) who
found that asthma hospitalisation rates are higher in Maori than in non-Maori,
despite asthma prevalence being similar in Maori and non-Maori
children.21
They conclude that the differences are likely to reflect differences in asthma
exacerbation and disease severity due to reduced access to asthma health
services.
Although there is considerable evidence for the existence of
significant disparities between Maori and non-Maori in socioeconomic status,
distribution of risk factors for disease, and access to healthcare, there is
less definitive research identifying the barriers and obstacles to good health
outcomes. It has been suggested that the younger age structure of the Maori
population, in comparison to the general population, may play a role in the
inequalities.22
Failure to account for these structural differences, when designing and
delivering services, may help explain why health promotion and education
strategies have had less impact on this ethnic group. Cost has been recognised
as a barrier to care with regard to diabetes and, given the socioeconomic data
previously discussed, it is likely that this obstacle plays a significant role
in other areas of Maori health
care.22,23
Although caution is required in attributing blame, the
Government reforms of the 1980s and early 1990s may also have had a
disproportionate effect on Maori. The sharp rise in Maori suicide rates may
suggest that Maori suffered considerably during this period.
In 1980, Maori men aged 24–44 years had suicide rates
22% less than non–Maori—but by 1999, the rates were 70% higher than
non-Maori.6
More recently, Malcolm (2003) has questioned whether health resources are
actually filtering down to where they are needed most and suggests that the
inverse care law (ie, those populations in greatest need are those least likely
to receive the services they need) remains a dominant feature of New
Zealand’s primary care
system.24
To summarise, the epidemiological data highlight several
important points in terms of healthcare policy and funding. First, Maori have
significantly lower life expectancy than non-Maori; the high mortality rates are
largely due to the burden of chronic disease. Second, Maori are over-represented
in socioeconomic-deprivation statistics; a measure highly correlated with
greater morbidity and mortality. Third, and most importantly in terms of
‘The Orewa Speech’, Maori have worse health outcomes independent of
socioeconomic measures. Finally, Maori suffer disproportionately from diseases
amenable to health service intervention.
Together, this evidence provides a compelling argument for
specific initiatives focused on improving Maori health outcomes and reducing
disparities. Contrary to the opinions of Dr Brash, current evidence identifies a
need for health policies to continue to directly target Maori and further, aim
to elucidate the barriers to care that presently exist.
The arguments above cite epidemiological evidence for
targeting Maori as an ethnic group. However, there are other grounds, the most
obvious of which is the Treaty of Waitangi.
The Treaty, signed in 1840, consists of three
articles:
It may be argued that the
current inequalities in health status between Maori and non-Maori violate this
contract and that the Crown has not provided equal protection and rights to
Maori compared with non-Maori. For example, Reid et al (2000) state that the
current disparities are a breach of the Treaty that sought to protect Maori from
marginalisation as a result of
colonisation.12
Durie (1989) notes that the deteriorating health of Maori
may actually have been the driving force behind a formal relationship between
the British Crown and
Maori.25
He states that a British protectorate was proposed in 1837 due to the
‘miserable’ condition of the Maori people, particularly their
‘high mortality rate’ resulting from ‘total European’
impact.
The Treaty of Waitangi represents the New Zealand
Government’s contractual obligation to explicitly ensure equitable
outcomes for Maori. Thus it offers a legal impetus for addressing the continuing
health needs of this specific ethnic group.
The Ministry of Health, amongst other Government bodies,
recognise these responsibilities and obligations. In terms of the New Zealand
Health
Strategy,26
recognition of the Treaty translates to the participation of Maori at all
levels, partnership with Maori in service delivery, and the protection and
improvement of Maori health status.
In conclusion, epidemiological data provide strong support
for specifically addressing Maori health need, whilst the Treaty of Waitangi
represents a contractual obligation on behalf of the New Zealand Government to
ensure equity of outcome for Maori. Underpinning both the epidemiological and
legal arguments are ethical principles. The central tenets of medicine, to
reduce suffering, improve and prolong the quality of life, should provide a
strong driving force to address these inequalities.
Health policies should be shaped by the four basic
principles of medical ethics: beneficence, non-maleficence, justice and
autonomy. The moral imperatives that form the very basis of the medical
profession should drive us (its practitioners and students) to strongly resist
any health policy that attempts to thwart the progress of Maori towards equality
in health status.
In essence, with regard to The Orewa Speech, Dr
Brash’s claims that Maori have the ‘upper hand’ remain
unsubstantiated. Unfortunately, these views may provide yet another threat to
Maori health.
Author information:
Cindy Towns, PhD Student, Department of Anatomy and Structural Biology; Nathan
Watkins, Medical Student; Arapera Salter,
Medical Student; Patricia Boyd, Medical Student, Dunedin School of
Medicine; Lianne Parkin, Lecturer in Epidemiology, Department of Social and
Preventive Medicine, University of Otago, Dunedin.
Correspondence:
Cindy Towns, C/- Department of Anatomy and Structural Biology, University of
Otago, Dunedin. Fax: 03 479 7254; email: cindy.towns@anatomy.otago.ac.nz
References:
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